¸ñÀû: During the initial phase of complete paralysis, complications are due to weakness of the affected muscles, but when recovery is partial, facial asymmetry and inability to refine fine movements can be caused either by prolonged paralysis or by the development of post-paralytic synkinesis. At such a phase of chronic facial paralysis, there has been observed different aspects of facial movement according to their pathogenesis. ¹æ¹ý:Four categorized pathogenesis of chronic facial paralysis could be found in our office. 1)group of patients with resection and anastomosis (n=15) 2)group of patients with chronic nerve compressive injury (n=13) 3)group of patients with severe Bells palsy (n=68) 4)group of patients with total resection without any reanimation surgery. (n=8). All of them (n=104) had chronic facial paralysis for more than 2 years from onset and were investigated severity of synkinesis, regression of muscular volume, latency and amplitude in EMG, and facial movement by HB grade system. °á°ú: Different aspect according to their pathogenesis can be found. Patients of first group had severe synkinesis without regression of muscular volume, HB grade 3.70.76. Patients of second group had mild synkinesis with minimal regression of muscular volume, HB grade 3.70.44, Patients of third group had moderate to severe synkinesis with moderate regression of muscular volume, HB grade 3.40.96. Patient of forth group had no synkinesis with profound regression of muscular volume, HB grade 5.70.34. °á·Ð:Clinical configuration of facial movement including severity of synkinesis, muscular regression, and facial weakness can be reflected by pathogenesis of facial nerve injury. Minute observation of facial movement is a crucial point for elucidating the pathogenesis of facial nerve paralysis and realizing any desire of the patients. |